The first reason behind postprandial dysmetabolism is the occurrence of elevated levels of glucose and fats in the bloodstream following the consumption of an unhealthy meal.
The extreme portions of glucose and lipids that pour into the blood following a excessive fats, high sugar meal causes a massive release of damaging free radicals and a cascade of pathological, tissue damaging events.
The body's own antioxidants are unable to cope with this deluge of free radicals and a state of oxidative stress ensues. Oxidative stress, in flip, causes the damaging processes that characterize postprandial dysmetabolism.
Traits of postprandial dysmetabolism:
- Inflammation: A general inflammatory course of occurs that not only aggravates the deposition of LDL (dangerous) cholesterol however increases the chances of atherosclerotic plaque rupture within the arteries thereby enhancing the danger for a heart assault or stroke.
- Endothelial dysfunction: The endothelium (or lining) of the arteries responds to oxidative stress by signaling the arterial wall to constrict, thereby increasing the risk of coronary heart assault and stroke.
- Thrombosis: The blood exhibits hypercoagulability - i.e it has a larger tendency to clot and form thromboses in the heart and brain.
- Increased sympathetic nerve activity: Stimulation of the sympathetic nervous system causes vasoconstriction and narrowing of the coronary and different arteries.
The traits of postpradial dysmetabolism all mix to substantially enhance the danger of cardiac and cerebral events both instantly after a meal and by way of long term atherosclerotic harm to the cardiovascular system.
It is important to word that it isn't the excessive ranges of glucose and fat per se that causes the damage. Relatively it is a state of oxidative stress resulting from the excessive ranges of glucose and fats that's at the root of the problem.
The prevention of free radical injury and oxidative stress by rising consumption of anti-oxidant wealthy foods similar to spices is likely one of the methods wherein we can stop the incidence of postprandial dysmetabolism.
Although eating substantial quantities of both animal and plant fats will trigger an increase in triglyceride ranges, not all kinds of fat or oils will cause oxidative stress. Research has proven that neither canola oil nor salmon oil trigger postprandial oxidative stress while animal fat and olive oil do certainly cause this pathological condition.
Does this mean that we've got to keep away from eating candy and oily foods utterly?
Although the sensible factor can be to considerably reduce the excessive consumption of those foods - and it will also assist us to reduce weight if we needed to do so - there are other methods to obviate the consequences of the damaging, postprandial dysmetabolic cascade.
While high blood glucose and triglyceride levels are the first cause of postprandial dysmetabolism it's the ensuing oxidative stress that truly causes damage to the arteries.
And there are effective ways to counter oxidative stress and free radical damage.
Prevention of Postprandial Dysmetabolism
- Eat a eating regimen excessive in minimally processed foods especially vegetables, fruit, whole grains, nuts, lean protein, vinegar, fish and canola oil.
- Lose excess weight and exercise regularly.
- Do not relaxation instantly after a meal. Analysis has shown that there is an elevated danger of heart attack in those who relaxation or sleep immediately after a meal. Gentle train after a meal helps to reduce excessive glucose and triglyceride levels and thus reduces the results of postprandial dysmetabolism.
- Spices: Embrace many various culinary herbs and spices in your diet. Spices have the highest antioxidant activity of all food varieties, include pure anti-inflammatory compounds and reverse many of the pathological processes associated with postprandial dysmetabolism: They scale back the fast absorption of fats and sugar from the intestine, management high blood sugar, have sturdy anti-inflammatory properties, assist dilate the arteries, inhibit the deposition of LDL (unhealthy) cholesterol in the arteries and reduce the danger of thrombus formation.
The invention of postprandial dysmetabolism has elevated our understanding of the essential position that the immediate results of a meal play within the causation of cardiovascular disease. However it has also provided us with simple, efficient measures that we are able to take to prevent this injurious condition from occurring.
0 comments:
Post a Comment